Thr50
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Home > Phosphorylation Site Page: > Thr50  -  RACK1 (rat)

Site Information
WKLTRDEtNyGIPQR   SwissProt Entrez-Gene
Blast this site against: NCBI  SwissProt  PDB 
Site Group ID: 23136904

In vivo Characterization
Methods used to characterize site in vivo:
immunoprecipitation ( 1 ) , mutation of modification site ( 1 ) , western blotting ( 1 )
Relevant cell line - cell type - tissue:
cerebral artery ( 1 ) , neuron ( 1 )

Upstream Regulation
Treatments:
deoxygenation ( 1 ) , dorsomorphin ( 1 ) , renal ischemia/reperfusion injury ( 1 )

Downstream Regulation
Effects of modification on RACK1:
intracellular localization ( 1 ) , molecular association, regulation ( 1 ) , protein degradation ( 1 )
Effects of modification on biological processes:
apoptosis, inhibited ( 1 ) , autophagy, induced ( 1 ) , translation, induced ( 1 )
Induce interaction with:
beclin 1 (human) ( 1 )

References 

1

Li X, et al. (2018) Loss of Ribosomal RACK1 (Receptor for Activated Protein Kinase C 1) Induced by Phosphorylation at T50 Alleviates Cerebral Ischemia-Reperfusion Injury in Rats. Stroke, STROKEAHA118022404
30580718   Curated Info