Jin K, et al. (2009) Mirk regulates the exit of colon cancer cells from quiescence. J Biol Chem 284, 22916-25 19542220

This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.

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T288-p - CCND1 (mouse) ▲

Modsite: AGLACtPtDVRDVDI SwissProt Entrez-Gene Orthologous residues CCND1 (human): T288‑p, CCND1 (mouse): T288‑p, CCND1 (rat): T288‑p Characterization Methods used to characterize site in vivo:  flow cytometry, mutation of modification site Disease tissue studied:  colorectal cancer, colorectal carcinoma Relevant cell lines - cell types - tissues:  HT-29 (intestinal) Cellular systems studied:  cell lines Species studied:  human Downstream Regulation Effect of modification (process):  cell cycle regulation