Curated Information
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Home > Curated Information Page > PubMed Id: 29988075
Kim SY, et al. (2018) Transient inhibition of p53 homologs protects ovarian function from two distinct apoptotic pathways triggered by anticancer therapies. Cell Death Differ 29988075
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.
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S160-p - p63 (mouse)
Modsite: SSTFDALsPsPAIPS SwissProt Entrez-Gene
Orthologous residues
p63 (human): S160‑p, p63 iso2 (human): S66‑p, p63 (mouse): S160‑p, p63 (rat): S160‑p
Characterization
Methods used to characterize site in vivo immunoassay, phospho-antibody, western blotting
Relevant cell lines - cell types - tissues:  ovarian
Cellular systems studied:  cell lines
Species studied:  human
Upstream Regulation
Treatments, proteins and their effect on site modification: 
Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes
cisplatin no change compared to control
ionizing_radiation increase X-ray 0.45gy

S162-p - p63 (mouse)
Modsite: TFDALsPsPAIPSNT SwissProt Entrez-Gene
Orthologous residues
p63 (human): S162‑p, p63 iso2 (human): S68‑p, p63 (mouse): S162‑p, p63 (rat): S162‑p
Characterization
Methods used to characterize site in vivo immunoassay, phospho-antibody, western blotting
Relevant cell lines - cell types - tissues:  ovarian
Cellular systems studied:  cell lines
Species studied:  human
Upstream Regulation
Treatments, proteins and their effect on site modification: 
Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes
cisplatin no change compared to control
ionizing_radiation increase X-ray 0.45gy