Sullivan JM, et al. (2016) Phosphorylation Regulates Id2 Degradation and Mediates the Proliferation of Neural Precursor Cells. Stem Cells 34, 1321-31 26756672

This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.

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S5-p - ID2 (human) ▲

Modsite: ___MKAFsPVRSVRk SwissProt Entrez-Gene Orthologous residues ID2 (human): S5‑p, ID2 (mouse): S5‑p, ID2 (rat): S5‑p Characterization Methods used to characterize site in vivo:  flow cytometry, mass spectrometry, mutation of modification site Relevant cell lines - cell types - tissues:  'stem, neural' Cellular systems studied:  cell lines Species studied:  human, mouse Downstream Regulation Effect of modification (function):  protein stabilization Effect of modification (process):  cell growth, inhibited

S14-p - ID2 (human) ▲

Modsite: VRSVRkNsLsDHSLG SwissProt Entrez-Gene Orthologous residues ID2 (human): S14‑p, ID2 (mouse): S14‑p, ID2 (rat): S14‑p Characterization Methods used to characterize site in vivo:  flow cytometry, mass spectrometry, mutation of modification site Relevant cell lines - cell types - tissues:  'stem, neural' Cellular systems studied:  cell lines Species studied:  human, mouse Downstream Regulation Effect of modification (function):  protein stabilization Effect of modification (process):  cell growth, inhibited

T27-p - ID2 (human) ▲

Modsite: LGISRSKtPVDDPMS SwissProt Entrez-Gene Orthologous residues ID2 (human): T27‑p, ID2 (mouse): T27‑p, ID2 (rat): T27‑p Characterization Methods used to characterize site in vivo:  flow cytometry, mass spectrometry, mutation of modification site Relevant cell lines - cell types - tissues:  'stem, neural' Cellular systems studied:  cell lines Species studied:  human, mouse Downstream Regulation Effect of modification (function):  protein stabilization Effect of modification (process):  cell growth, inhibited