Curated Information
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Home > Curated Information Page > PubMed Id: 26453379
Keppler SJ, et al. (2015) Wiskott-Aldrich Syndrome Interacting Protein Deficiency Uncovers the Role of the Co-receptor CD19 as a Generic Hub for PI3 Kinase Signaling in B Cells. Immunity 43, 660-73 26453379
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.
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S478-p - WIPF1 (mouse)
Modsite: RNESRSGsNRRERGA SwissProt Entrez-Gene
Orthologous residues
WIPF1 (human): S488‑p, WIPF1 iso2 (human): , WIPF1 (mouse): S478‑p, WIPF1 (rat): S472‑p
Methods used to characterize site in vivo phospho-antibody, western blotting
Disease tissue studied:  lymphoma, B cell lymphoma
Relevant cell lines - cell types - tissues:  WEHI-231 (B lymphocyte)
Cellular systems studied:  cell lines
Species studied:  mouse
Upstream Regulation
Treatments, proteins and their effect on site modification: 
Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes
antigenic stimulation increase BCR crosslinking