Nicholson TB, et al. (2013) A hypomorphic lsd1 allele results in heart development defects in mice. PLoS One 8, e60913 23637775

This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.

Click on the protein name to open the protein page, and on the RSD number to open the site page.

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S840-p - CDH1 (mouse) ▲

Modsite: LVFDYEGsGsEAAsL SwissProt Entrez-Gene Orthologous residues CDH1 (human): S838‑p, CDH1 (mouse): S840‑p, CDH1 (rat): S842‑p Characterization Methods used to characterize site in vivo:  immunoassay, phospho-antibody, western blotting Relevant cell lines - cell types - tissues:  heart Cellular systems studied:  tissue Species studied:  mouse Upstream Regulation Treatments, proteins and their effect on site modification:  Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes increase in hypomorphic hearts of Aof2 2lox/2lox homozygous mice

S842-p - CDH1 (mouse) ▲

Modsite: FDYEGsGsEAAsLss SwissProt Entrez-Gene Orthologous residues CDH1 (human): S840‑p, CDH1 (mouse): S842‑p, CDH1 (rat): S844‑p Characterization Methods used to characterize site in vivo:  immunoassay, phospho-antibody, western blotting Relevant cell lines - cell types - tissues:  heart Cellular systems studied:  tissue Species studied:  mouse Upstream Regulation Treatments, proteins and their effect on site modification:  Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes increase in hypomorphic hearts of Aof2 2lox/2lox homozygous mice