Curated Information
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Home > Curated Information Page > PubMed Id: 28972129
Li DP, Zhou JJ, Zhang J, Pan HL (2017) CaMKII Regulates Synaptic NMDA Receptor Activity of Hypothalamic Presympathetic Neurons and Sympathetic Outflow in Hypertension. J Neurosci 37, 10690-10699 28972129
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.
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S1303-p - NMDAR2B (rat)
Modsite: NKLRRQHsyDtFVDL SwissProt Entrez-Gene
Orthologous residues
NMDAR2B (human): S1303‑p, NMDAR2B (mouse): S1303‑p, NMDAR2B (rat): S1303‑p
Characterization
Methods used to characterize site in vivo phospho-antibody, western blotting
Relevant cell lines - cell types - tissues:  'brain, cerebral cortex', 'brain, hippocampus', 'brain, hypothalamus'
Cellular systems studied:  tissue
Species studied:  rat
Comments:  hypothalamic paraventricular nucleus (PVN); frontal cortex
Associated Diseases
Diseases Alterations Comments
hypertension increased increased phosphorylation in the hypothalamic paraventricular nucleus (PVN), but not in the hippocampus and frontal cortex,