Curated Information
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Kreider-Letterman G, et al. (2023) ARHGAP17 regulates the spatiotemporal activity of Cdc42 at invadopodia. J Cell Biol 222 36571786
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2015, 43:D512-20). To learn more about the scope of PhosphoSitePlus®, click here.
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S702-p - ARHGAP17 (human)
Modsite: LsAPRRYsssLsPIQ SwissProt Entrez-Gene
Orthologous residues
ARHGAP17 (human): S702‑p, ARHGAP17 iso2 (human): S624‑p, ARHGAP17 iso5 (human): S702‑p, ARHGAP17 (mouse): S698‑p, ARHGAP17 iso2 (mouse): S620‑p, ARHGAP17 (rat): S710‑p
Characterization
Methods used to characterize site in vivo mutation of modification site
Disease tissue studied:  breast cancer
Relevant cell lines - cell types - tissues:  SUM159 (breast cell)
Cellular systems studied:  cell lines
Species studied:  human
Downstream Regulation
Effect of modification (function):  intracellular localization
Comments:  regulates invadopodia disassembly