Curated Information
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Curated Information Page
PubMed Id: 17145504 
This page summarizes selected information from the article referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Hsieh H, et al. (2006) AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine loss. Neuron 52, 831-43 17145504
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S880-p - GluR2 (rat)
Orthologous residues
GluR2 (human): S880‑p, GluR2 iso2 (human): S880‑p, GluR2 (mouse): S880‑p, GluR2 iso3 (mouse): , GluR2 (rat): S880‑p
Characterization
 Methods used to characterize site in vivo phospho-antibody
 Relevant cell lines - cell types - tissues:  'neuron, cortical'-brain
 Cellular systems studied:  primary cultured cells
 Species studied:  rat
Upstream Regulation
 Treatments, proteins and their effect on site modification: 
Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes
beta-amyloid 42 increase
Downstream Regulation
 Effect of modification (function):  receptor internalization, altered


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