Curated Information
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Curated Information Page
PubMed Id: 16600858 
Hu HJ, et al. (2006) The kv4.2 potassium channel subunit is required for pain plasticity. Neuron 50, 89-100 16600858
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
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S616-p - Kv4.2 (mouse)
Orthologous residues
Kv4.2 (human): S616‑p, Kv4.2 (mouse): S616‑p, Kv4.2 (rat): S616‑p
Characterization
 Methods used to characterize site in vivo mutation of modification site
 Relevant cell lines - cell types - tissues:  'neuron, dorsal horn'-spinal cord
 Cellular systems studied:  primary cultured cells
 Species studied:  mouse
Upstream Regulation
 Potential in vivo enzymes for site: 
Type Enzyme Evidence Notes
KINASE ERK1 (mouse)
KINASE ERK2 (mouse)
Downstream Regulation
 Effect of modification (function):  activity, induced
 Comments:  modulates Kv4.2-mediated A-type currents in spinal cord dorsal horn neurons


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