Curated Information
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Curated Information Page
PubMed Id: 9694798 
Partanen J, Schwartz L, Rossant J (1998) Opposite phenotypes of hypomorphic and Y766 phosphorylation site mutations reveal a function for Fgfr1 in anteroposterior patterning of mouse embryos. Genes Dev 12, 2332-44 9694798
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Information from this record has been curated, but not yet edited in PhosphoSitePlus® and may be incomplete.
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Y766-p - FGFR1 (mouse)
Orthologous residues
FGFR1 (human): Y766‑p, FGFR1 iso14 (human): Y675‑p, FGFR1 iso21 (human): Y797‑p, FGFR1 (mouse): Y766‑p, FGFR1 (rat): Y673‑p, FGFR1 iso2 (rat): Y766‑p
 Methods used to characterize site in vivo mutation of modification site
 Relevant cell lines - cell types - tissues:  embryo [FGFR1 (mouse), transgenic]
 Cellular systems studied:  tissue
 Species studied:  mouse
Downstream Regulation
 Effect of modification (process):  cell growth, altered, transcription, altered
 Comments:  Y766F mutation causes embryonic transformations in vertebral column.

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