Curated Information
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Curated Information Page
PubMed Id: 19074970 
This page summarizes selected information from the article referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Yarotskyy V, Gao G, Peterson BZ, Elmslie KS (2009) The Timothy syndrome mutation of cardiac CaV1.2 (L-type) channels: multiple altered gating mechanisms and pharmacological restoration of inactivation. J Physiol 587, 551-65 19074970
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S439-p - CACNA1C (rabbit)
Orthologous residues
CACNA1C (human): S409‑p, CACNA1C iso12 (human): S409‑p, CACNA1C (mouse): S409‑p, CACNA1C iso5 (mouse): S439‑p, CACNA1C (rat): S439‑p, CACNA1C (rabbit): S439‑p, CACNA1C iso4 (rabbit): S409‑p, CACNA1C (guinea pig): S438‑p
Characterization
 Methods used to characterize site in vivo mutation of modification site
 Relevant cell lines - cell types - tissues:  293 (epithelial)
 Cellular systems studied:  cell lines
 Species studied:  human


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