Curated Information
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Curated Information Page
PubMed Id: 18223294 
This page summarizes selected information from the article referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Kaiserova K, Tang XL, Srivastava S, Bhatnagar A (2008) Role of nitric oxide in regulating aldose reductase activation in the ischemic heart. J Biol Chem 283, 9101-12 18223294
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S1176-p - eNOS (rat)
Orthologous residues
eNOS (human): S1177‑p, eNOS (mouse): S1176‑p, eNOS (rat): S1176‑p, eNOS (rabbit): S1183‑p, eNOS (pig): S1179‑p, eNOS (cow): S1179‑p
Characterization
 Methods used to characterize site in vivo phospho-antibody, western blotting
 Relevant cell lines - cell types - tissues:  heart
 Cellular systems studied:  tissue
 Species studied:  rat
Upstream Regulation
 Treatments, proteins and their effect on site modification: 
Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes
bradykinin increase
insulin increase
ischemia increase
bradykinin ischemia augment treatment-induced increase
insulin ischemia augment treatment-induced increase
LY294002 ischemia inhibit treatment-induced increase


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