Curated Information
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Curated Information Page
PubMed Id: 14741104 
This page summarizes selected information from the article referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Gaudillière B, et al. (2004) A CaMKII-NeuroD signaling pathway specifies dendritic morphogenesis. Neuron 41, 229-41 14741104
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S336-p - NEUROD1 (rat)
Orthologous residues
NEUROD1 (human): S335‑p, NEUROD1 (mouse): S336‑p, NEUROD1 (rat): S336‑p
Characterization
 Methods used to characterize site in vivo electrophoretic mobility shift, mutation of modification site, phospho-antibody, western blotting
 Relevant cell lines - cell types - tissues:  'neuron, cerebellar granule'-brain
 Cellular systems studied:  primary cultured cells
 Species studied:  rat
 Enzymes shown to modify site in vitro
Type Enzyme
KINASE CAMK2A (rat)
Upstream Regulation
 Potential in vivo enzymes for site: 
Type Enzyme Evidence Notes
KINASE CAMK2A (rat) pharmacological inhibitor of upstream enzyme, genetic knockout/knockin of upstream enzyme
 Treatments, proteins and their effect on site modification: 
Treatments Referenced Treatments Manipulated Protein Referenced Protein Effect Notes
depolarization increase
KN-62 depolarization inhibit treatment-induced increase
nimodipine depolarization inhibit treatment-induced increase
U0126 depolarization no effect upon treatment-induced increase
Downstream Regulation
 Effect of modification (process):  cell growth, altered


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