Curated Information
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Curated Information Page
PubMed Id: 20154723 
Li Z, et al. (2010) An HDAC1-binding domain within FATS bridges p21 turnover to radiation-induced tumorigenesis. Oncogene 29, 2659-71 20154723
This page summarizes selected information from the record referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Information from this record has been curated, but not yet edited in PhosphoSitePlus® and may be incomplete.
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K141-ac - p21Cip1 (human)
Orthologous residues
p21Cip1 (human): K141‑ac, p21Cip1 (mouse): K136‑ac, p21Cip1 (dog): K109‑ac

K154-ac - p21Cip1 (human)
Orthologous residues
p21Cip1 (human): K154‑ac, p21Cip1 (mouse): K149‑ac, p21Cip1 (dog): K122‑ac

K161-ac - p21Cip1 (human)
Orthologous residues
p21Cip1 (human): K161‑ac, p21Cip1 (mouse): K156‑ac, p21Cip1 (dog): K129‑ac

K163-ac - p21Cip1 (human)
Orthologous residues
p21Cip1 (human): K163‑ac, p21Cip1 (mouse): K158‑ac, p21Cip1 (dog): K131‑ac

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