Curated Information
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Curated Information Page
PubMed Id: 9872318 
This page summarizes selected information from the article referenced above and curated into PhosphoSitePlus®, a comprehensive online resource for the study of protein post-translational modifications (NAR, 2012,40:D261-70). To learn more about the scope of PhosphoSitePlus®, click here.
Schroeder BC, Kubisch C, Stein V, Jentsch TJ (1998) Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy. Nature 396, 687-90 9872318
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S52-p - Kv7.2 (human) ▲
Orthologous residues
Kv7.2 (human): S52‑p, Kv7.2 iso3 (human): S52‑p, Kv7.2 (mouse): S52‑p, Kv7.2 iso3 (mouse): S52‑p, Kv7.2 iso12 (mouse): S52‑p, Kv7.2 (rat): S52‑p
Characterization
 Methods used to characterize site in vivo:  mutation of modification site
 Disease tissue studied:  Status epilepticus
 Relevant cell lines - cell types - tissues:  293 (epithelial)
 Cellular systems studied:  cell lines
 Species studied:  human
Downstream Regulation
 Effect of modification (function):  activation


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